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Cholesterol accumulation-induced impairment of AKT signaling in LPS-stimulated macrophages play a dispensable role in suppressing HIF-1α-dependent glycolysis
View ORCID ProfileKenneth K.Y. Ting, View ORCID ProfileMyron I. Cybulsky
doi: https://doi.org/10.1101/2024.09.01.610717
Kenneth K.Y. Ting
1Department of Immunology, University of Toronto, Toronto, ON M5S 1A8, Canada
2Toronto General Hospital Research Institute, University Health Network, Toronto, ON M5G 1L7, Canada
Myron I. Cybulsky
1Department of Immunology, University of Toronto, Toronto, ON M5S 1A8, Canada
2Toronto General Hospital Research Institute, University Health Network, Toronto, ON M5G 1L7, Canada
3Department of Laboratory Medicine and Pathobiology, University of Toronto, Toronto, ON M5S 1A8, Canada
4Peter Munk Cardiac Centre, University Health Network, Toronto, ON M5G 2N2, Canada
Posted September 03, 2024.
Cholesterol accumulation-induced impairment of AKT signaling in LPS-stimulated macrophages play a dispensable role in suppressing HIF-1α-dependent glycolysis
Kenneth K.Y. Ting, Myron I. Cybulsky
bioRxiv 2024.09.01.610717; doi: https://doi.org/10.1101/2024.09.01.610717
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