Article Information
History
- October 16, 2017.
Copyright
The copyright holder for this preprint is the author/funder, who has granted bioRxiv a license to display the preprint in perpetuity. All rights reserved. No reuse allowed without permission.
Author Information
- Ann Rancourt1,4,
- Sébastien Dufresne25,
- Guillaume St-Pierre1,
- Julie-Christine Lévesque3,
- Haruka Nakamura1,
- Yodai Kikuchi1,
- Masahiko S. Satoh4,
- Jérôme Frenette2 and
- Sachiko Sato1,3,#
- 1Glycobiology and Bioimaging Laboratory, Research Centre for Infectious Diseases, Faculty of Medicine, Laval University, Quebec City, Canada
- 2Research Centre of CHU de Québec, and Department of Rehabilitation, Faculty of Medicine, Laval University, Quebec City, Canada
- 3Bioimaging Platform, Research Centre of CHU de Québec, Quebec City, Canada
- 4Laboratory of DNA Damage Responses and Bioimaging, Research Centre of CHU de Québec, Faculty of Medicine, Laval University, Quebec City, Canada
- ↵*Correspondence should be addressed to S.S., Sachiko Sato, Research Centre for Infectious Diseases, RC-709 CHUL-CHU de Québec Research Centre, 2705 boul, Laurier, Québec, QC, Canada G1V 4G2, Tel:1-418-525-4444 extension 48647, Fax:1-418-654-2715, email sachiko.sato{at}crchul.ulaval.ca
Posted October 16, 2017.
Galectin-3 and N-acetylglucosamine promote myogenesis and improve skeletal muscle function in the mdx model of Duchenne muscular dystrophy
