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HMGA1 zebrafish co-orthologue hmga1b can modulate p53-dependent cellular responses but is unable to control the alternative splicing of psen1
View ORCID ProfileSeyed Hani Moussavi Nik, View ORCID ProfileMorgan Newman, View ORCID ProfileAmanda Lumsden, Tanya Jayne, View ORCID ProfileMichael Lardelli
doi: https://doi.org/10.1101/241927
Seyed Hani Moussavi Nik
1Alzheimer’s Disease Genetics Laboratory, Center for Molecular Pathology, School of Biological Sciences, University of Adelaide, Adelaide, South Australia, Australia
Morgan Newman
1Alzheimer’s Disease Genetics Laboratory, Center for Molecular Pathology, School of Biological Sciences, University of Adelaide, Adelaide, South Australia, Australia
Amanda Lumsden
2College of Medicine and Public Health, and Center for Neuroscience, Flinders University, Adelaide, South Australia, Australia
Tanya Jayne
1Alzheimer’s Disease Genetics Laboratory, Center for Molecular Pathology, School of Biological Sciences, University of Adelaide, Adelaide, South Australia, Australia
Michael Lardelli
1Alzheimer’s Disease Genetics Laboratory, Center for Molecular Pathology, School of Biological Sciences, University of Adelaide, Adelaide, South Australia, Australia

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Posted January 02, 2018.
HMGA1 zebrafish co-orthologue hmga1b can modulate p53-dependent cellular responses but is unable to control the alternative splicing of psen1
Seyed Hani Moussavi Nik, Morgan Newman, Amanda Lumsden, Tanya Jayne, Michael Lardelli
bioRxiv 241927; doi: https://doi.org/10.1101/241927
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