Article Information
History
- July 6, 2018.
Copyright
The copyright holder for this preprint is the author/funder, who has granted bioRxiv a license to display the preprint in perpetuity. It is made available under a CC-BY 4.0 International license.
Author Information
- Claudio Toma1,2,
- Kerrie D. Pierce1,
- Alex D. Shaw1,2,
- Anna Heath1,
- Philip B. Mitchell3,4,
- Peter R. Schofield1,2 and
- Janice M. Fullerton1,2
- 1Neuroscience Research Australia, Sydney, Australia
- 2School of Medical Sciences, University of New South Wales, Sydney, Australia
- 3School of Psychiatry, University of New South Wales, Sydney, Australia
- 4Black Dog Institute, Prince of Wales Hospital, Sydney, Australia
- Corresponding author:
Claudio Toma, Neuroscience Research Australia, Margarete Ainsworth Building, Barker Street, Randwick NSW 2031, Sydney, Australia. Tel.: (+61) 29399 1890; Email: c.toma{at}neura.edu.au
Posted July 06, 2018.
Comprehensive cross-disorder analyses of CNTNAP2 suggest it is unlikely to be a primary risk gene for psychiatric disorders
