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Hypoxia-induced switch in SNAT2/SLC38A2 regulation generates endocrine-resistance in breast cancer
Matteo Morotti, Esther Bridges, Alessandro Valli, Hani Choudhry, Helen Sheldon, Simon Wigfield, Nicki Gray, Dylan Jones, Eugene J. Teoh, Wei-Chen Cheng, Simon Lord, Syed Haider, Alan McIntyre, Deborah C. I. Goberdhan, Francesca Buffa, Adrian L. Harris
doi: https://doi.org/10.1101/384628
Matteo Morotti
1Hypoxia and Angiogenesis Group, Cancer Research UK Molecular Oncology Laboratories, Weatherall Institute of Molecular Medicine, Department of Oncology, University of Oxford, Oxford, United Kingdom
Esther Bridges
1Hypoxia and Angiogenesis Group, Cancer Research UK Molecular Oncology Laboratories, Weatherall Institute of Molecular Medicine, Department of Oncology, University of Oxford, Oxford, United Kingdom
Alessandro Valli
1Hypoxia and Angiogenesis Group, Cancer Research UK Molecular Oncology Laboratories, Weatherall Institute of Molecular Medicine, Department of Oncology, University of Oxford, Oxford, United Kingdom
Hani Choudhry
2Department of Biochemistry, Faculty of Science, King Abdulaziz University, Jeddah, Saudi Arabia
Helen Sheldon
1Hypoxia and Angiogenesis Group, Cancer Research UK Molecular Oncology Laboratories, Weatherall Institute of Molecular Medicine, Department of Oncology, University of Oxford, Oxford, United Kingdom
Simon Wigfield
1Hypoxia and Angiogenesis Group, Cancer Research UK Molecular Oncology Laboratories, Weatherall Institute of Molecular Medicine, Department of Oncology, University of Oxford, Oxford, United Kingdom
Nicki Gray
3Computational Biology Research Group, Weatherall Institute of Molecular Medicine, John Radcliffe Hospital, University of Oxford, Oxford, United Kingdom
Dylan Jones
1Hypoxia and Angiogenesis Group, Cancer Research UK Molecular Oncology Laboratories, Weatherall Institute of Molecular Medicine, Department of Oncology, University of Oxford, Oxford, United Kingdom
Eugene J. Teoh
1Hypoxia and Angiogenesis Group, Cancer Research UK Molecular Oncology Laboratories, Weatherall Institute of Molecular Medicine, Department of Oncology, University of Oxford, Oxford, United Kingdom
Wei-Chen Cheng
4Computational Biology and Integrative Genomics, Department of Oncology, University of Oxford, Oxford, United Kingdom
Simon Lord
1Hypoxia and Angiogenesis Group, Cancer Research UK Molecular Oncology Laboratories, Weatherall Institute of Molecular Medicine, Department of Oncology, University of Oxford, Oxford, United Kingdom
Syed Haider
1Hypoxia and Angiogenesis Group, Cancer Research UK Molecular Oncology Laboratories, Weatherall Institute of Molecular Medicine, Department of Oncology, University of Oxford, Oxford, United Kingdom
3Computational Biology Research Group, Weatherall Institute of Molecular Medicine, John Radcliffe Hospital, University of Oxford, Oxford, United Kingdom
5The Breast Cancer Now Toby Robins Research Centre, Division of Breast Cancer Research, The Institute of Cancer Research, London, United Kingdom
Alan McIntyre
6Cancer Biology, Division of Cancer and Stem Cells, The University of Nottingham, Nottingham, United Kingdom
Deborah C. I. Goberdhan
7Department of Physiology, Anatomy and Genetics, Le Gros Clark Building, University of Oxford, Oxford, United Kingdom
Francesca Buffa
4Computational Biology and Integrative Genomics, Department of Oncology, University of Oxford, Oxford, United Kingdom
Adrian L. Harris
1Hypoxia and Angiogenesis Group, Cancer Research UK Molecular Oncology Laboratories, Weatherall Institute of Molecular Medicine, Department of Oncology, University of Oxford, Oxford, United Kingdom
Article usage
Posted August 03, 2018.
Hypoxia-induced switch in SNAT2/SLC38A2 regulation generates endocrine-resistance in breast cancer
Matteo Morotti, Esther Bridges, Alessandro Valli, Hani Choudhry, Helen Sheldon, Simon Wigfield, Nicki Gray, Dylan Jones, Eugene J. Teoh, Wei-Chen Cheng, Simon Lord, Syed Haider, Alan McIntyre, Deborah C. I. Goberdhan, Francesca Buffa, Adrian L. Harris
bioRxiv 384628; doi: https://doi.org/10.1101/384628
Hypoxia-induced switch in SNAT2/SLC38A2 regulation generates endocrine-resistance in breast cancer
Matteo Morotti, Esther Bridges, Alessandro Valli, Hani Choudhry, Helen Sheldon, Simon Wigfield, Nicki Gray, Dylan Jones, Eugene J. Teoh, Wei-Chen Cheng, Simon Lord, Syed Haider, Alan McIntyre, Deborah C. I. Goberdhan, Francesca Buffa, Adrian L. Harris
bioRxiv 384628; doi: https://doi.org/10.1101/384628
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