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Cyclin D-Cdk4,6 drives cell cycle progression via the retinoblastoma protein’s C-terminal helix
Benjamin R. Topacio, Evgeny Zatulovskiy, Sandra Cristea, Shicong Xie, Carrie S. Tambo, Seth M. Rubin, Julien Sage, Mardo Kõivomägi, Jan M. Skotheim
doi: https://doi.org/10.1101/397448
Benjamin R. Topacio
1Department of Biology, Stanford University, Stanford, CA 94305, USA
Evgeny Zatulovskiy
1Department of Biology, Stanford University, Stanford, CA 94305, USA
Sandra Cristea
2Department of Pediatrics, Stanford University School of Medicine, Stanford, CA 94305, USA
3Department of Genetics, Stanford University School of Medicine, Stanford, CA 94305, USA
Shicong Xie
1Department of Biology, Stanford University, Stanford, CA 94305, USA
Carrie S. Tambo
4Department of Chemistry and Biochemistry, University of California, Santa Cruz, CA 95064, USA
Seth M. Rubin
4Department of Chemistry and Biochemistry, University of California, Santa Cruz, CA 95064, USA
Julien Sage
2Department of Pediatrics, Stanford University School of Medicine, Stanford, CA 94305, USA
3Department of Genetics, Stanford University School of Medicine, Stanford, CA 94305, USA
Mardo Kõivomägi
1Department of Biology, Stanford University, Stanford, CA 94305, USA
Jan M. Skotheim
1Department of Biology, Stanford University, Stanford, CA 94305, USA
Posted August 21, 2018.
Cyclin D-Cdk4,6 drives cell cycle progression via the retinoblastoma protein’s C-terminal helix
Benjamin R. Topacio, Evgeny Zatulovskiy, Sandra Cristea, Shicong Xie, Carrie S. Tambo, Seth M. Rubin, Julien Sage, Mardo Kõivomägi, Jan M. Skotheim
bioRxiv 397448; doi: https://doi.org/10.1101/397448
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