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Mitotic CDK promotes replisome disassembly, fork breakage, and complex DNA rearrangements
Lin Deng, R. Alex. Wu, Olga V. Kochenova, David Pellman, Johannes C. Walter
doi: https://doi.org/10.1101/428433
Lin Deng
1Department of Pediatric Oncology, Dana-Farber Cancer Institute, Boston, MA 02215, USA
2Department of Cell Biology, Harvard Medical School, Boston, MA 02115, USA
3Department of Biological Chemistry and Molecular Pharmacology, Harvard Medical School, Boston, MA 02115, USA
R. Alex. Wu
3Department of Biological Chemistry and Molecular Pharmacology, Harvard Medical School, Boston, MA 02115, USA
Olga V. Kochenova
3Department of Biological Chemistry and Molecular Pharmacology, Harvard Medical School, Boston, MA 02115, USA
David Pellman
1Department of Pediatric Oncology, Dana-Farber Cancer Institute, Boston, MA 02215, USA
2Department of Cell Biology, Harvard Medical School, Boston, MA 02115, USA
4Broad Institute of MIT and Harvard, Cambridge, MA 02142, USA
5Howard Hughes Medical Institute, Boston, MA 02115, USA
Johannes C. Walter
3Department of Biological Chemistry and Molecular Pharmacology, Harvard Medical School, Boston, MA 02115, USA
5Howard Hughes Medical Institute, Boston, MA 02115, USA
Article usage
Posted September 26, 2018.
Mitotic CDK promotes replisome disassembly, fork breakage, and complex DNA rearrangements
Lin Deng, R. Alex. Wu, Olga V. Kochenova, David Pellman, Johannes C. Walter
bioRxiv 428433; doi: https://doi.org/10.1101/428433
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