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Loss of Rap1 supports recombination-based telomere maintenance independent of RNA-DNA hybrids in fission yeast
Yan Hu, Henrietta W. Bennett, Na Liu, Martin Moravec, Jessica F. Williams, View ORCID ProfileClaus M. Azzalin, Megan C. King
doi: https://doi.org/10.1101/458968
Yan Hu
1Department of Cell Biology, Yale School of Medicine, 333 Cedar Street, New Haven, CT, 06520-8002, USA
Henrietta W. Bennett
1Department of Cell Biology, Yale School of Medicine, 333 Cedar Street, New Haven, CT, 06520-8002, USA
3Current affiliation: Stanford University School of Medicine, Stanford, California, 943055441, USA
Na Liu
1Department of Cell Biology, Yale School of Medicine, 333 Cedar Street, New Haven, CT, 06520-8002, USA
Martin Moravec
2Institute of Biochemistry (IBC), Eidgenössische Technische Hochschule Zürich (ETHZ), Zürich, CH-8093, Switzerland
Jessica F. Williams
1Department of Cell Biology, Yale School of Medicine, 333 Cedar Street, New Haven, CT, 06520-8002, USA
Claus M. Azzalin
2Institute of Biochemistry (IBC), Eidgenössische Technische Hochschule Zürich (ETHZ), Zürich, CH-8093, Switzerland
4Current affiliation: Instituto de Medicina Moleculare, Lisboa, Portugal
Megan C. King
1Department of Cell Biology, Yale School of Medicine, 333 Cedar Street, New Haven, CT, 06520-8002, USA
Posted November 01, 2018.
Loss of Rap1 supports recombination-based telomere maintenance independent of RNA-DNA hybrids in fission yeast
Yan Hu, Henrietta W. Bennett, Na Liu, Martin Moravec, Jessica F. Williams, Claus M. Azzalin, Megan C. King
bioRxiv 458968; doi: https://doi.org/10.1101/458968
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