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A Drosophila model of neuronal ceroid lipofuscinosis CLN4 reveals a hypermorphic gain of function mechanism

Elliot Imler, Jin Sang Pyon, Selina Kindelay, Yong-quan Zhang, Sreeganga S. Chandra, Konrad E. Zinsmaier
doi: https://doi.org/10.1101/579771
Elliot Imler
1Graduate Interdisciplinary Program in Neuroscience, University of Arizona, Tucson, AZ 85721, USA;
5Department of Neuroscience, University of Arizona, Tucson, AZ 85721, USA.
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Jin Sang Pyon
2Undergraduate Program in Neuroscience and Cognitive Science Department of Molecular, University of Arizona, Tucson, AZ 85721, USA;
5Department of Neuroscience, University of Arizona, Tucson, AZ 85721, USA.
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Selina Kindelay
2Undergraduate Program in Neuroscience and Cognitive Science Department of Molecular, University of Arizona, Tucson, AZ 85721, USA;
5Department of Neuroscience, University of Arizona, Tucson, AZ 85721, USA.
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Yong-quan Zhang
3Departments of Neuroscience and Neurology, Yale University, New Haven, CT 06536, USA;
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Sreeganga S. Chandra
3Departments of Neuroscience and Neurology, Yale University, New Haven, CT 06536, USA;
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Konrad E. Zinsmaier
4Department of Molecular and Cellular Biology, University of Arizona, Tucson, AZ 85721, USA;
5Department of Neuroscience, University of Arizona, Tucson, AZ 85721, USA.
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Article Information

doi 
https://doi.org/10.1101/579771
History 
  • March 15, 2019.
Copyright 
The copyright holder for this preprint is the author/funder, who has granted bioRxiv a license to display the preprint in perpetuity. It is made available under a CC-BY-NC-ND 4.0 International license.

Author Information

  1. Elliot Imler1,5,
  2. Jin Sang Pyon2,5,
  3. Selina Kindelay2,5,
  4. Yong-quan Zhang3,
  5. Sreeganga S. Chandra3 and
  6. Konrad E. Zinsmaier4,5
  1. 1Graduate Interdisciplinary Program in Neuroscience, University of Arizona, Tucson, AZ 85721, USA;
  2. 2Undergraduate Program in Neuroscience and Cognitive Science Department of Molecular, University of Arizona, Tucson, AZ 85721, USA;
  3. 3Departments of Neuroscience and Neurology, Yale University, New Haven, CT 06536, USA;
  4. 4Department of Molecular and Cellular Biology, University of Arizona, Tucson, AZ 85721, USA;
  5. 5Department of Neuroscience, University of Arizona, Tucson, AZ 85721, USA.
  1. Correspondence to: Konrad E. Zinsmaier: kez4{at}email.arizona.edu.
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Posted March 15, 2019.
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A Drosophila model of neuronal ceroid lipofuscinosis CLN4 reveals a hypermorphic gain of function mechanism
Elliot Imler, Jin Sang Pyon, Selina Kindelay, Yong-quan Zhang, Sreeganga S. Chandra, Konrad E. Zinsmaier
bioRxiv 579771; doi: https://doi.org/10.1101/579771
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A Drosophila model of neuronal ceroid lipofuscinosis CLN4 reveals a hypermorphic gain of function mechanism
Elliot Imler, Jin Sang Pyon, Selina Kindelay, Yong-quan Zhang, Sreeganga S. Chandra, Konrad E. Zinsmaier
bioRxiv 579771; doi: https://doi.org/10.1101/579771

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