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History
- March 15, 2019.
Copyright
The copyright holder for this preprint is the author/funder, who has granted bioRxiv a license to display the preprint in perpetuity. It is made available under a CC-BY-NC-ND 4.0 International license.
Author Information
- Elliot Imler1,5,
- Jin Sang Pyon2,5,
- Selina Kindelay2,5,
- Yong-quan Zhang3,
- Sreeganga S. Chandra3 and
- Konrad E. Zinsmaier4,5
- 1Graduate Interdisciplinary Program in Neuroscience, University of Arizona, Tucson, AZ 85721, USA;
- 2Undergraduate Program in Neuroscience and Cognitive Science Department of Molecular, University of Arizona, Tucson, AZ 85721, USA;
- 3Departments of Neuroscience and Neurology, Yale University, New Haven, CT 06536, USA;
- 4Department of Molecular and Cellular Biology, University of Arizona, Tucson, AZ 85721, USA;
- 5Department of Neuroscience, University of Arizona, Tucson, AZ 85721, USA.
- Correspondence to: Konrad E. Zinsmaier: kez4{at}email.arizona.edu.
Posted March 15, 2019.
A Drosophila model of neuronal ceroid lipofuscinosis CLN4 reveals a hypermorphic gain of function mechanism
