Article Information
History
- May 2, 2019.
Copyright
The copyright holder for this preprint is the author/funder, who has granted bioRxiv a license to display the preprint in perpetuity. It is made available under a CC-BY-NC-ND 4.0 International license.
Author Information
- Aaron Gordon, PhD1,
- Annika Grønborg-Forsingdal, PhD2,3,
- Ib Vestergaard Klewe, PhD2,
- Jacob Nielsen, PhD,
- Michael Didriksen, PhD,
- Thomas Werge, PhD4,5,6 and
- Daniel Geschwind, MD PhD7,8,9,*
- 1Department of Neurology, University of California Los Angeles, Los Angeles, CA
- 2Division of Synaptic Transmission, H. Lundbeck A/S, Valby, Denmark
- 3Institute of Biological Psychiatry, Mental Health Services Capital Region of Denmark, Copenhagen, Denmark
- 4Lundbeck Foundation Initiative for Integrative Psychiatric Research, iPSYCH, Copenhagen, Denmark
- 5Department of Clinical Medicine, Faculty of Health and Medical Sciences, University of Copenhagen, Copenhagen, Denmark
- 6Lundbeck Foundation GeoGenetics Centre, Natural History Museum of Denmark, University of Copenhagen, 1350 Copenhagen, Denmark
- 7Program in Neurobehavioral Genetics, Semel Institute, David Geffen School of Medicine, University of California, Los Angeles, CA
- 8Department of Neurology, Center for Autism Research and Treatment, Semel Institute, David Geffen School of Medicine, University of California, Los Angeles, CA
- 9Department of Human Genetics, David Geffen School of Medicine, University of California, Los Angeles, CA
- ↵*Corresponding author email: Daniel H Geschwind, 2506 Gonda Bldg, 695 Charles E. Young Dr. South, Los Angeles, CA 90095-1761, Telephone: +1-310-794-6570, Fax: +1-310-267-2401, dhg{at}mednet.ucla.edu
Posted May 02, 2019.
Transcriptomic networks implicate neuronal energetic abnormalities in three mouse models harboring autism and schizophrenia-associated mutations
