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Mutually exclusive autism mutations point to the circadian clock and PI3K signaling pathways

Hannah Manning, Brian J. O’Roak, Özgün Babur
doi: https://doi.org/10.1101/653527
Hannah Manning
1Computational Biology Program, Oregon Health & Science University, Portland, OR 97239
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Brian J. O’Roak
2Department of Molecular and Medical Genetics, Oregon Health & Science University, Portland, OR 97239
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  • For correspondence: ozgunbabur@gmail.com
Özgün Babur
1Computational Biology Program, Oregon Health & Science University, Portland, OR 97239
2Department of Molecular and Medical Genetics, Oregon Health & Science University, Portland, OR 97239
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Article Information

doi 
https://doi.org/10.1101/653527
History 
  • May 30, 2019.
Copyright 
The copyright holder for this preprint is the author/funder, who has granted bioRxiv a license to display the preprint in perpetuity. It is made available under a CC-BY 4.0 International license.

Author Information

  1. Hannah Manning1,
  2. Brian J. O’Roak2,* and
  3. Özgün Babur1,2,*
  1. 1Computational Biology Program, Oregon Health & Science University, Portland, OR 97239
  2. 2Department of Molecular and Medical Genetics, Oregon Health & Science University, Portland, OR 97239
  1. ↵*Corresponding author; email: ozgunbabur{at}gmail.com
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Posted May 30, 2019.
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Mutually exclusive autism mutations point to the circadian clock and PI3K signaling pathways
Hannah Manning, Brian J. O’Roak, Özgün Babur
bioRxiv 653527; doi: https://doi.org/10.1101/653527
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Mutually exclusive autism mutations point to the circadian clock and PI3K signaling pathways
Hannah Manning, Brian J. O’Roak, Özgün Babur
bioRxiv 653527; doi: https://doi.org/10.1101/653527

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