Article Information
History
- October 25, 2019.
Article Versions
- Version 1 (June 3, 2019 - 22:02).
- You are viewing Version 2, the most recent version of this article.
Copyright
The copyright holder for this preprint is the author/funder, who has granted bioRxiv a license to display the preprint in perpetuity. This article is a US Government work. It is not subject to copyright under 17 USC 105 and is also made available for use under a CC0 license.
Author Information
- 1Graduate Program in Molecular and Cellular Biology, Uniformed Services University of the Health Sciences, F. Edward Hébert School of Medicine, Bethesda, MD 20814
- 2Division of Cardiovascular Medicine, Department of Medicine, The University of Virginia School of Medicine, Charlottesville, VA 22908
- 3Department of Anatomy, Physiology, and Genetics, Uniformed Services University of the Health Sciences, F. Edward Hébert School of Medicine, Bethesda, MD 20814
- ↵*Corresponding Author:
Jeremy T. Smyth, Uniformed Services University of the Health Sciences, 4301 Jones Bridge Rd, Bethesda, MD 20814, Tel: (301) 295-5879, Email: jeremy.smyth{at}usuhs.edu
Posted October 25, 2019.
Suppression of store-operated calcium entry causes dilated cardiomyopathy of the Drosophila heart
