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A gatekeeping function of the replicative polymerase controls pathway choice in the resolution of lesion-stalled replisomes
Seungwoo Chang, Karel Naiman, Elizabeth S. Thrall, James E. Kath, Slobodan Jergic, Nicholas Dixon, Robert P. Fuchs, Joseph J Loparo
doi: https://doi.org/10.1101/676486
Seungwoo Chang
1Department of Biological Chemistry and Molecular Pharmacology, Harvard Medical School, Boston, USA
Karel Naiman
2Team DNA Damage Tolerance, Cancer Research Center of Marseille (CRCM), CNRS, UMR7258, Marseille, F-13009, France
Elizabeth S. Thrall
1Department of Biological Chemistry and Molecular Pharmacology, Harvard Medical School, Boston, USA
James E. Kath
1Department of Biological Chemistry and Molecular Pharmacology, Harvard Medical School, Boston, USA
Slobodan Jergic
3School of Chemistry, University of Wollongong, Wollongong, New South Wales, Australia
Nicholas Dixon
3School of Chemistry, University of Wollongong, Wollongong, New South Wales, Australia
Robert P. Fuchs
2Team DNA Damage Tolerance, Cancer Research Center of Marseille (CRCM), CNRS, UMR7258, Marseille, F-13009, France
Joseph J Loparo
1Department of Biological Chemistry and Molecular Pharmacology, Harvard Medical School, Boston, USA
Article usage
Posted June 21, 2019.
A gatekeeping function of the replicative polymerase controls pathway choice in the resolution of lesion-stalled replisomes
Seungwoo Chang, Karel Naiman, Elizabeth S. Thrall, James E. Kath, Slobodan Jergic, Nicholas Dixon, Robert P. Fuchs, Joseph J Loparo
bioRxiv 676486; doi: https://doi.org/10.1101/676486
A gatekeeping function of the replicative polymerase controls pathway choice in the resolution of lesion-stalled replisomes
Seungwoo Chang, Karel Naiman, Elizabeth S. Thrall, James E. Kath, Slobodan Jergic, Nicholas Dixon, Robert P. Fuchs, Joseph J Loparo
bioRxiv 676486; doi: https://doi.org/10.1101/676486
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