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The most common RNF43 mutant G659Vfs41 is fully functional in inhibiting Wnt signaling and unlikely to play a role in tumorigenesis
Jianghua Tu, Soohyun Park, Wangsheng Yu, Sheng Zhang, Ling Wu, Kendra Carmon, Qingyun J. Liu
doi: https://doi.org/10.1101/711382
Jianghua Tu
1Texas Therapeutics Institute and The Brown Foundation Institute of Molecular Medicine, The University of Texas Health Science Center at Houston, 1825 Pressler St., Houston Texas, USA
Soohyun Park
1Texas Therapeutics Institute and The Brown Foundation Institute of Molecular Medicine, The University of Texas Health Science Center at Houston, 1825 Pressler St., Houston Texas, USA
Wangsheng Yu
1Texas Therapeutics Institute and The Brown Foundation Institute of Molecular Medicine, The University of Texas Health Science Center at Houston, 1825 Pressler St., Houston Texas, USA
Sheng Zhang
1Texas Therapeutics Institute and The Brown Foundation Institute of Molecular Medicine, The University of Texas Health Science Center at Houston, 1825 Pressler St., Houston Texas, USA
Ling Wu
1Texas Therapeutics Institute and The Brown Foundation Institute of Molecular Medicine, The University of Texas Health Science Center at Houston, 1825 Pressler St., Houston Texas, USA
Kendra Carmon
1Texas Therapeutics Institute and The Brown Foundation Institute of Molecular Medicine, The University of Texas Health Science Center at Houston, 1825 Pressler St., Houston Texas, USA
Qingyun J. Liu
1Texas Therapeutics Institute and The Brown Foundation Institute of Molecular Medicine, The University of Texas Health Science Center at Houston, 1825 Pressler St., Houston Texas, USA
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Posted July 22, 2019.
The most common RNF43 mutant G659Vfs41 is fully functional in inhibiting Wnt signaling and unlikely to play a role in tumorigenesis
Jianghua Tu, Soohyun Park, Wangsheng Yu, Sheng Zhang, Ling Wu, Kendra Carmon, Qingyun J. Liu
bioRxiv 711382; doi: https://doi.org/10.1101/711382
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