Article Information
History
- July 30, 2019.
Copyright
The copyright holder for this preprint is the author/funder, who has granted bioRxiv a license to display the preprint in perpetuity. It is made available under a CC-BY-NC-ND 4.0 International license.
Author Information
- Nathalie Faggianelli-Conrozier1,2,3,4,8,
- Aikaterini Polyzou5,6,8,
- Renee Chow1,2,3,4,
- Stéphane Roth1,2,3,4,
- Eirini Trompouki5,7,* and
- Julien Vermot1,2,3,4,*
- 1Institut de Génétique et de Biologie Moléculaire et Cellulaire, 67404 Illkirch, France
- 2Centre National de la Recherche Scientifique, UMR7104, 67404 Illkirch, France
- 3Institut National de la Santé et de la Recherche Médicale, U964, 67404 Illkirch, France
- 4Université de Strasbourg, 67404 Illkirch, France
- 5Department of Cellular and Molecular Immunology, Max Planck Institute of Immunobiology and Epigenetics, 51 Stübeweg, 79108, Freiburg, Germany
- 6Faculty of Biology, University of Freiburg, Schänzlestraße 1, 79104, Freiburg, Germany
- 7CIBSS-Centre for Integrative Biological Signalling Studies, Freiburg, Germany
- ↵*Correspondence: trompouki{at}ie-freiburg.mpg.de, julien{at}igbmc.fr
↵8 These authors contributed equally
Posted July 30, 2019.
Complementary functions of the mechanosensitive factors egr1, klf2b and klf2a instruct the endocardial program
