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High-dose oncogenic PIK3CA drives constitutive cellular stemness through self-sustained TGFβ pathway activation
View ORCID ProfileRalitsa R. Madsen, View ORCID ProfileJames Longden, Rachel G. Knox, View ORCID ProfileXavier Robin, Franziska Völlmy, Kenneth G. Macleod, Larissa Moniz, View ORCID ProfileNeil O. Carragher, View ORCID ProfileNicholas McGranahan, View ORCID ProfileRune Linding, View ORCID ProfileBart Vanhaesebroeck, View ORCID ProfileRobert K. Semple
doi: https://doi.org/10.1101/753830
Ralitsa R. Madsen
1Centre for Cardiovascular Science, Queen’s Medical Research Institute, University of Edinburgh, Edinburgh, UK
2Metabolic Research Laboratories, Wellcome Trust-MRC Institute of Metabolic Science, University of Cambridge, Cambridge, UK
3The National Institute for Health Research Cambridge Biomedical Research Centre, Cambridge, UK
James Longden
4Biotech Research and Innovation Centre, University of Copenhagen, Copenhagen, Denmark
5Humboldt-Universität zu Berlin, Berlin, Germany
Rachel G. Knox
2Metabolic Research Laboratories, Wellcome Trust-MRC Institute of Metabolic Science, University of Cambridge, Cambridge, UK
3The National Institute for Health Research Cambridge Biomedical Research Centre, Cambridge, UK
Xavier Robin
4Biotech Research and Innovation Centre, University of Copenhagen, Copenhagen, Denmark
Franziska Völlmy
4Biotech Research and Innovation Centre, University of Copenhagen, Copenhagen, Denmark
Kenneth G. Macleod
6Edinburgh Cancer Research UK Centre, Institute of Genetics and Molecular Medicine, University of Edinburgh, Western General Hospital, Crewe Road South, Edinburgh, UK
Larissa Moniz
7University College London Cancer Institute, Paul O’Gorman Building, University College London, London, UK
Neil O. Carragher
6Edinburgh Cancer Research UK Centre, Institute of Genetics and Molecular Medicine, University of Edinburgh, Western General Hospital, Crewe Road South, Edinburgh, UK
Nicholas McGranahan
7University College London Cancer Institute, Paul O’Gorman Building, University College London, London, UK
Rune Linding
4Biotech Research and Innovation Centre, University of Copenhagen, Copenhagen, Denmark
5Humboldt-Universität zu Berlin, Berlin, Germany
Bart Vanhaesebroeck
7University College London Cancer Institute, Paul O’Gorman Building, University College London, London, UK
Robert K. Semple
1Centre for Cardiovascular Science, Queen’s Medical Research Institute, University of Edinburgh, Edinburgh, UK
2Metabolic Research Laboratories, Wellcome Trust-MRC Institute of Metabolic Science, University of Cambridge, Cambridge, UK
3The National Institute for Health Research Cambridge Biomedical Research Centre, Cambridge, UK
Posted September 01, 2019.
High-dose oncogenic PIK3CA drives constitutive cellular stemness through self-sustained TGFβ pathway activation
Ralitsa R. Madsen, James Longden, Rachel G. Knox, Xavier Robin, Franziska Völlmy, Kenneth G. Macleod, Larissa Moniz, Neil O. Carragher, Nicholas McGranahan, Rune Linding, Bart Vanhaesebroeck, Robert K. Semple
bioRxiv 753830; doi: https://doi.org/10.1101/753830
High-dose oncogenic PIK3CA drives constitutive cellular stemness through self-sustained TGFβ pathway activation
Ralitsa R. Madsen, James Longden, Rachel G. Knox, Xavier Robin, Franziska Völlmy, Kenneth G. Macleod, Larissa Moniz, Neil O. Carragher, Nicholas McGranahan, Rune Linding, Bart Vanhaesebroeck, Robert K. Semple
bioRxiv 753830; doi: https://doi.org/10.1101/753830
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