Article Information
History
- October 17, 2019.
Copyright
The copyright holder for this preprint is the author/funder, who has granted bioRxiv a license to display the preprint in perpetuity. It is made available under a CC-BY-NC-ND 4.0 International license.
Author Information
- Anja Armache1,#,
- Shuang Yang2,#,
- Lexi E Robbins3,
- Ceyda Durmaz3,
- Andrew W Daman3,
- Jin Q Jeong3,
- Alexia Martínez de Paz3,
- Arjun Ravishankar3,
- Tanja Arslan4,
- Shu Lin5,
- Tanya Panchenko1,6,
- Benjamin A. Garcia5,
- Sandra B. Hake4,7,
- Haitao Li2,*,
- C. David Allis1,* and
- Steven Z. Josefowicz3,*
- 1The Rockefeller University, Laboratory of Chromatin Biology and Epigenetics, New York, NY, US 10065
- 2Center for Life Sciences, Tsinghua University, Beijing, China 100084
- 3Weill Cornell Medicine, Laboratory of Epigenetics and Immunity, New York, NY, US 10065
- 4Adolf-Butenandt Institute, Ludwig-Maximilians University, Munich, Germany, 80336
- 5Epigenetics Institute, Department of Biochemistry and Biophysics, University of Pennsylvania, Philadelphia, PA, US 19104
- 6Perlmutter Cancer Center, New York University Langone Medical Center, New York, NY 10016
- 7Institute for Genetics, Justus-Liebig-University Giessen, Germany 35392
- ↵*Corresponding Authors szj2001{at}med.cornell.edu, alliscd{at}rockefeller.edu, lht{at}tsinghua.edu.cn
↵# equal contribution
Posted October 17, 2019.
Phosphorylation of the ancestral histone variant H3.3 amplifies stimulation-induced transcription
