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Fungal derived 15-keto-prostaglandin E2 and host proliferator-activated receptor gamma (PPAR-γ) promote C. neoformans growth during infection
Robert J. Evans, Sarah Needs, Ewa Bielska, Robin C. May, Stephen A. Renshaw, Simon A. Johnston
doi: https://doi.org/10.1101/113167
Robert J. Evans
1Bateson Centre, Firth Court, University of Sheffield, S10 2TN, UK.
2Department of Infection, Immunity and Cardiovascular Disease, Medical School, University of Sheffield, S10 2RX, UK.
Sarah Needs
3Institute of Microbiology and Infection, School of Biosciences, University of Birmingham, Birmingham, B15 2TT, UK.
Ewa Bielska
3Institute of Microbiology and Infection, School of Biosciences, University of Birmingham, Birmingham, B15 2TT, UK.
Robin C. May
3Institute of Microbiology and Infection, School of Biosciences, University of Birmingham, Birmingham, B15 2TT, UK.
4NIHR Surgical Reconstruction and Microbiology Research Centre, University Hospitals of Birmingham NHS Foundation Trust, Queen Elizabeth Hospital, Birmingham, UK.
Stephen A. Renshaw
1Bateson Centre, Firth Court, University of Sheffield, S10 2TN, UK.
2Department of Infection, Immunity and Cardiovascular Disease, Medical School, University of Sheffield, S10 2RX, UK.
Simon A. Johnston
1Bateson Centre, Firth Court, University of Sheffield, S10 2TN, UK.
2Department of Infection, Immunity and Cardiovascular Disease, Medical School, University of Sheffield, S10 2RX, UK.
Article usage
Posted March 02, 2017.
Fungal derived 15-keto-prostaglandin E2 and host proliferator-activated receptor gamma (PPAR-γ) promote C. neoformans growth during infection
Robert J. Evans, Sarah Needs, Ewa Bielska, Robin C. May, Stephen A. Renshaw, Simon A. Johnston
bioRxiv 113167; doi: https://doi.org/10.1101/113167
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