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Combined inhibition of apoptosis and necrosis promotes transient neuroprotection of retinal ganglion cells and partial-axon regeneration after optic nerve damage
Maki Kayama, Kumiko Omura, Yusuke Murakami, Edith Reshef, Aristomenis Thanos, Yuki Morizane, Andrea Giani, Toru Nakazawa, Joan W. Miller, Larry Benowitz, View ORCID ProfileDemetrios G. Vavvas
doi: https://doi.org/10.1101/357566
Maki Kayama
aRetina Service, Angiogenesis Laboratory, Massachusetts Eye and Ear Infirmary
bDepartment of Ophthalmology, Harvard Medical School, Boston, MA 02114, USA
Kumiko Omura
dLaboratory for Neuroscience Research in Neurosurgery and F.M. Kirby Neurobiology Center, Boston Children’s Hospital, 300 Longwood Ave., Boston MA 02115 USA.
eDepartment of Neurosurgery, Harvard Medical School, Boston MA 02115 USA.
Yusuke Murakami
aRetina Service, Angiogenesis Laboratory, Massachusetts Eye and Ear Infirmary
Edith Reshef
aRetina Service, Angiogenesis Laboratory, Massachusetts Eye and Ear Infirmary
dLaboratory for Neuroscience Research in Neurosurgery and F.M. Kirby Neurobiology Center, Boston Children’s Hospital, 300 Longwood Ave., Boston MA 02115 USA.
Aristomenis Thanos
aRetina Service, Angiogenesis Laboratory, Massachusetts Eye and Ear Infirmary
bDepartment of Ophthalmology, Harvard Medical School, Boston, MA 02114, USA
Yuki Morizane
aRetina Service, Angiogenesis Laboratory, Massachusetts Eye and Ear Infirmary
bDepartment of Ophthalmology, Harvard Medical School, Boston, MA 02114, USA
Andrea Giani
aRetina Service, Angiogenesis Laboratory, Massachusetts Eye and Ear Infirmary
bDepartment of Ophthalmology, Harvard Medical School, Boston, MA 02114, USA
Toru Nakazawa
aRetina Service, Angiogenesis Laboratory, Massachusetts Eye and Ear Infirmary
bDepartment of Ophthalmology, Harvard Medical School, Boston, MA 02114, USA
cDepartment of Ophthalmology, Tohoku Graduate University School of Medicine, Miyagi, 980-8574, JAPAN
Joan W. Miller
aRetina Service, Angiogenesis Laboratory, Massachusetts Eye and Ear Infirmary
bDepartment of Ophthalmology, Harvard Medical School, Boston, MA 02114, USA
Larry Benowitz
dLaboratory for Neuroscience Research in Neurosurgery and F.M. Kirby Neurobiology Center, Boston Children’s Hospital, 300 Longwood Ave., Boston MA 02115 USA.
eDepartment of Neurosurgery, Harvard Medical School, Boston MA 02115 USA.
Demetrios G. Vavvas
aRetina Service, Angiogenesis Laboratory, Massachusetts Eye and Ear Infirmary
bDepartment of Ophthalmology, Harvard Medical School, Boston, MA 02114, USA
Article usage
Posted June 27, 2018.
Combined inhibition of apoptosis and necrosis promotes transient neuroprotection of retinal ganglion cells and partial-axon regeneration after optic nerve damage
Maki Kayama, Kumiko Omura, Yusuke Murakami, Edith Reshef, Aristomenis Thanos, Yuki Morizane, Andrea Giani, Toru Nakazawa, Joan W. Miller, Larry Benowitz, Demetrios G. Vavvas
bioRxiv 357566; doi: https://doi.org/10.1101/357566
Combined inhibition of apoptosis and necrosis promotes transient neuroprotection of retinal ganglion cells and partial-axon regeneration after optic nerve damage
Maki Kayama, Kumiko Omura, Yusuke Murakami, Edith Reshef, Aristomenis Thanos, Yuki Morizane, Andrea Giani, Toru Nakazawa, Joan W. Miller, Larry Benowitz, Demetrios G. Vavvas
bioRxiv 357566; doi: https://doi.org/10.1101/357566
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