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A symmetric toggle switch explains the onset of random X inactivation in different mammals
Verena Mutzel, Ikuhiro Okamoto, Ilona Dunkel, Mitinori Saitou, Luca Giorgetti, Edith Heard, View ORCID ProfileEdda G. Schulz
doi: https://doi.org/10.1101/204909
Verena Mutzel
1Regulatory Networks in Stem Cells, Otto-Warburg-Laboratorium, Max Planck Institute for molecular Genetics, 14195 Berlin, Germany
Ikuhiro Okamoto
2Department of Anatomy and Cell Biology, Graduate School of Medicine, Kyoto University, 606-8501, Japan; JST, ERATO, Kyoto 606-8501, Japan
Ilona Dunkel
1Regulatory Networks in Stem Cells, Otto-Warburg-Laboratorium, Max Planck Institute for molecular Genetics, 14195 Berlin, Germany
Mitinori Saitou
3Department of Anatomy and Cell Biology, Graduate School of Medicine, Kyoto University, 606-8501, Japan; JST, ERATO, Kyoto 606-8501, Japan; Center for iPS Cell Research and Application, Kyoto University, Kyoto 606-8507, Japan; Institute for Integrated Cell-Material Sciences, Kyoto University, Kyoto 606-8501, Japan
Luca Giorgetti
4Friedrich Miescher Institute for Biomedical Research, Basel, CH-4058, Switzerland
Edith Heard
5Institut Curie, PSL Research University, CNRS UMR3215, INSERM U934, Paris, France
Edda G. Schulz
1Regulatory Networks in Stem Cells, Otto-Warburg-Laboratorium, Max Planck Institute for molecular Genetics, 14195 Berlin, Germany
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Posted October 09, 2018.
A symmetric toggle switch explains the onset of random X inactivation in different mammals
Verena Mutzel, Ikuhiro Okamoto, Ilona Dunkel, Mitinori Saitou, Luca Giorgetti, Edith Heard, Edda G. Schulz
bioRxiv 204909; doi: https://doi.org/10.1101/204909
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