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A stress response that allows highly mutated eukaryotic cells to survive and proliferate
Rebecca A. Zabinsky, Jonathan Mares, Richard She, Michelle K. Zeman, Thomas R. Silvers, Daniel F. Jarosz
doi: https://doi.org/10.1101/515460
Rebecca A. Zabinsky
1Department of Chemical and Systems Biology, Stanford University, 269 Campus Drive, Stanford, CA 94305
Jonathan Mares
1Department of Chemical and Systems Biology, Stanford University, 269 Campus Drive, Stanford, CA 94305
Richard She
1Department of Chemical and Systems Biology, Stanford University, 269 Campus Drive, Stanford, CA 94305
Michelle K. Zeman
1Department of Chemical and Systems Biology, Stanford University, 269 Campus Drive, Stanford, CA 94305
Thomas R. Silvers
2Cancer Biology Program, Stanford University, 269 Campus Drive, Stanford, CA 94305
Daniel F. Jarosz
1Department of Chemical and Systems Biology, Stanford University, 269 Campus Drive, Stanford, CA 94305
2Cancer Biology Program, Stanford University, 269 Campus Drive, Stanford, CA 94305
3Department of Developmental Biology, Stanford University, 269 Campus Drive, Stanford, CA 94305
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Posted January 09, 2019.
A stress response that allows highly mutated eukaryotic cells to survive and proliferate
Rebecca A. Zabinsky, Jonathan Mares, Richard She, Michelle K. Zeman, Thomas R. Silvers, Daniel F. Jarosz
bioRxiv 515460; doi: https://doi.org/10.1101/515460
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