Trends in Microbiology
Volume 4, Issue 4, April 1996, Pages 129-131
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Sleepless in Bologna: transmission of fatal familial insomia

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  • Cited by (23)

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    • Prions and the Immune System: A Journey Through Gut, Spleen, and Nerves

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      Reintroduction of Prnp by transgenesis—even in a shortened, redacted form—restores infectibility and prion replication in Prnpo/o mice (Fischer et al., 1996; Shmerling et al., 1998; Supattapone et al., 1999; Flechsig et al., 2000). In addition, all familial cases of human TSEs are characterized by Prnp mutations (Aguzzi and Weissmann, 1996a; Prusiner et al., 1998). Informational nucleic acids of >50 nucleotides in length do not participate in prion infectivity (Kellings et al., 1993; Riesner et al., 1993), but shorter non-coding oligonucleotides have not been formally excluded—a fact that may have some relevance in view of the surprising discoveries related to RNA-mediated gene silencing.

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