User profiles for Akiki Marie

Rose Marie Akiki

Medical University of South Carolina
Verified email at musc.edu
Cited by 8

[HTML][HTML] Cacna1b alternative splicing impacts excitatory neurotransmission and is linked to behavioral responses to aversive stimuli

A Bunda, B LaCarubba, M Bertolino, M Akiki, K Bath… - Molecular brain, 2019 - Springer
Presynaptic Ca V 2.2 channels control calcium entry that triggers neurotransmitter release
at both central and peripheral synapses. The Cacna1b gene encodes the α1-pore forming …

Tissue‐ and cell‐specific expression of a splice variant in the II‐III cytoplasmic loop of Cacna1b

A Bunda, B LaCarubba, M Akiki, A Andrade - FEBS Open Bio, 2019 - Wiley Online Library
Presynaptic Ca V 2.2 (N‐type) channels are fundamental for transmitter release across the
nervous system. The gene encoding Ca V 2.2 channels, Cacna1b, contains alternatively …

NPAS4 in the medial prefrontal cortex mediates chronic social defeat stress-induced anhedonia-like behavior and reductions in excitatory synapses

…, S Berto, J Kumar, RG Cornbrooks, RM Akiki… - Elife, 2023 - elifesciences.org
Chronic stress can produce reward system deficits (ie, anhedonia) and other common
symptoms associated with depressive disorders, as well as neural circuit hypofunction in the …

Developmental and cell-specific expression of Cacna1d splice variants

LC Brianna, B Alexandra, S Kitty, S Hannah, A Marie… - bioRxiv, 2019 - biorxiv.org
Ca V 1.3 is an L-type voltage-gated calcium channel implicated in several functions
including gene expression, pacemaking activity, and neurotransmitter release. The gene that …

Tissue and cell-type specific expression of a splice variant in the II-III cytoplasmic loop of Cacna1b

B Alexandra, LC Brianna, A Marie, A Arturo - bioRxiv, 2019 - biorxiv.org
Presynaptic Ca V 2.2 (N-type) channels are fundamental for transmitter release across the
nervous system. The gene encoding Ca V 2.2 channels, Cacna1b, contains alternatively …

NPAS4 supports drug-cue associations and relapse-like behavior through regulation of the cell type-specific activation balance in the nucleus accumbens

…, E Tsvetkov, BM Siemsen, KK Snyder, RM Akiki… - BioRxiv, 2022 - biorxiv.org
Use of addictive substances creates powerful drug-cue associations that often trigger relapse.
Drug seeking is gated in the nucleus accumbens (NAc) by competing activation of D1 …

[PDF][PDF] NPAS4 in the medial prefrontal cortex mediates chronic social defeat stress-induced anhedonia and dendritic spine loss

…, S Berto, J Kumar, RG Cornbrooks, RM Akiki… - bioRxiv, 2021 - researchgate.net
Chronic stress can produce reward system deficits (ie anhedonia) and other common
symptoms associated with depressive disorders, as well as neural circuit hypofunction in the …

Non-coding enhancer RNA regulates NPAS4 in the mPFC to control chronic stress-induced anhedonia-like behavior

…, S Berto, J Kumar, RG Cornbrooks, RM Akiki… - bioRxiv, 2021 - biorxiv.org
Background Chronic stress can produce reward system deficits (i.e. anhedonia) and other
common symptoms associated with depressive disorders, as well as neural circuit …

Comparison of two delayed strategies for renal replacement therapy initiation for severe acute kidney injury (AKIKI 2): a multicentre, open-label, randomised …

S Gaudry, D Hajage, L Martin-Lefevre, S Lebbah… - The Lancet, 2021 - thelancet.com
Background Delaying renal replacement therapy (RRT) for some time in critically ill patients
with severe acute kidney injury and no severe complication is safe and allows optimisation …

Influence of local porosity and local permeability on the performances of a polymer electrolyte membrane fuel cell

T Akiki, W Charon, MC Iltchev, G Accary… - Journal of Power Sources, 2010 - Elsevier
In the literature, many models and studies focused on the steady-state aspect of fuel cell
systems while their dynamic transient behavior is still a wide area of research. In the present …