Background The biological mechanisms involved in inflammatory response to air pollution
are not clearly understood. Objective In this study we assessed the association of short-term …

In response to mounting concerns about the endocrine‐disrupting influence of environmental
chemicals on human health, this epidemiological study was initiated to test the hypothesis …

Background: Early-life exposures to tobacco smoke and some dietary factors have been
identified to induce epigenetic changes in genes involved in allergy and asthma development. …

BACKGROUND: Assessment of the adverse effects of oxidative stress related to air pollution
is limited by the lack of biological markers of dose to the lungs. OBJECTIVE: We evaluated …

Background Taffic-related air pollution has been related to adverse respiratory outcomes;
however, there is still uncertainty concerning the type of vehicle emission causing most …

Epigenetic regulation of imprinted genes is regarded as a highly plausible explanation for
linking dietary exposures in early life with the onset of diseases during childhood and …

Introduction Air pollutant exposure has been associated with an increase in inflammatory
markers and a decline in lung function in asthmatic children. Several studies suggest that …

Background: Air pollution is a problem, especially in developing countries. We examined the
association between personal exposure to particulate matter with an aerodynamic diameter …

Asthma is a complex disease with sex-specific differences in prevalence. Candidate gene
studies have suggested that genotype-by-sex interaction effects on asthma risk exist, but this …

Background Prenatal exposure to environmental pollutants such as mold, lead, pesticides,
tobacco, and air pollutants has been suggested to impair cognitive development. Evidence is …