[HTML][HTML] Alzheimer's genetic risk factor FERMT2 (Kindlin-2) controls axonal growth and synaptic plasticity in an APP-dependent manner

F Eysert, A Coulon, E Boscher, AC Vreulx, A Flaig… - Molecular …, 2021 - nature.com
Although APP metabolism is being intensively investigated, a large fraction of its modulators
is yet to be characterized. In this context, we combined two genome-wide high-content …

Correction to: Alzheimer's genetic risk factor FERMT2 (Kindlin-2) controls axonal growth and synaptic plasticity in an APP-dependent manner

F Eysert, A Coulon, E Boscher, AC Vreulx, A Flaig… - Molecular …, 2021 - nature.com
APP-dependent manner, written by Fanny Eysert, Audrey Coulon, Emmanuelle Boscher,
Anaїs-Camille Vreulx, Amandine Flaig, Tiago Mendes, Sandrine Hughes, Benjamin Grenier-…

Alzheimer's genetic risk factor FERMT2 (Kindlin-2) controls axonal growth and synaptic plasticity in an APP-dependent manner

F Eysert, A Coulon, E Boscher, AC Vreulx, A Flaig… - bioRxiv, 2019 - biorxiv.org
Although APP metabolism is being intensively investigated, a large fraction of its modulators
are yet to be characterized. In this context, we combined two genome-wide high-content …

[PDF][PDF] Alzheimer's genetic risk factor FERMT2 (Kindlin-2) is regulated by risk allele-binding of miR-4504 and leads to synaptic dysfunction in an APP-dependent …

F Eysert, A Coulon, E Boscher, AC Vreulx, A Flaig… - researchgate.net
The pathophysiological roles of Alzheimer’s disease (AD) genetic risk factors are often
unknown. In this study, we show that FERMT2 (or Kindlin-2) downregulation by miRNAs …

[PDF][PDF] Alzheimer's genetic risk factor FERMT2 (Kindlin-2) controls axonal growth and synaptic plasticity in an APP-dependent manner

A Flaig, T Mendes, S Hughes, B Grenier-Boley… - 2020 - hal.science
Although APP metabolism is being intensively investigated, a large fraction of its modulators
is yet to be characterized. In this context, we combined two genome-wide high-content …