User profiles for Ashutosh Prince

Ashutosh Prince

Postdoctoral Scholar, Cleveland State University
Verified email at csuohio.edu
Cited by 188

[HTML][HTML] Lipid-II independent antimicrobial mechanism of nisin depends on its crowding and degree of oligomerization

A Prince, P Sandhu, P Ror, E Dash, S Sharma… - Scientific reports, 2016 - nature.com
Nisin inhibits bacterial growth by generating pores in cell membrane and interrupting cell-wall
biosynthesis through specific lipid II interaction. However, the role of the hinge region and …

Attenuation of neuroblastoma cell growth by nisin is mediated by modulation of phase behavior and enhanced cell membrane fluidity

A Prince, A Tiwari, P Ror, P Sandhu, J Roy… - Physical Chemistry …, 2019 - pubs.rsc.org
Antimicrobial peptides have been attracting significant attention as potential anti-cancer
therapeutic agents in recent times. Yet most antimicrobial peptides seem to possess cytotoxic …

Passive membrane penetration by ZnO nanoparticles is driven by the interplay of electrostatic and phase boundary conditions

A Tiwari, A Prince, M Arakha, S Jha, M Saleem - Nanoscale, 2018 - pubs.rsc.org
The internalization of nanoparticles through the biological membrane is of immense importance
for biomedical applications. A fundamental understanding of the lipid specificity and the …

Perturbations of Cellular Model Membranes Induced by Membrane Interacting Model Peptide, Protein and Proteoliposome

A Prince - 2020 - ethesis.nitrkl.ac.in
The cell membrane interface constantly encounters a highly crowded environment of
membrane interacting biomolecular entities present, extracellularly and intracellularly, that both …

[PDF][PDF] Impavido attenuates inflammation, reduces atherosclerosis, and alters gut microbiota in hyperlipidemic mice

…, K Neupane, MR Khan, E Opoku, T Nunn, A Prince… - Iscience, 2023 - cell.com
Impavido (Miltefosine) is an FDA-approved drug for treating leishmaniasis and primary amebic
meningoencephalitis. We have shown previously that Miltefosine increased cholesterol …

Osteoblast‐derived giant plasma membrane vesicles induce osteogenic differentiation of human mesenchymal stem cells

…, T Agarwal, D Mukherjee, A Prince… - Advanced …, 2018 - Wiley Online Library
Exosome‐mediated differentiation of human mesenchymal stem cell (hMSC) has opened
up a new possibility for the directed osteogenic differentiation of hMSC by the osteoblast‐…

Corrigendum: lipid-II independent antimicrobial mechanism of nisin depends on its crowding and degree of oligomerization

A Prince, P Sandhu, P Ror, E Dash, S Sharma… - Scientific …, 2017 - ncbi.nlm.nih.gov
Ashutosh Prince, Padmani Sandhu, Pankaj Ror, Eva Dash, Shingarika Sharma,
Manoranjan Arakha, Suman Jha, Yusuf Akhter, and Mohammed Saleem …

Disulfiram reduces atherosclerosis and enhances efferocytosis, autophagy, and atheroprotective gut microbiota in hyperlipidemic mice

CA Traughber, K Timinski, A Prince, N Bhandari… - bioRxiv, 2023 - ncbi.nlm.nih.gov
Pyroptosis executor Gasdermin (GsdmD) promotes atherosclerosis in mice and humans.
Disulfiram (DSF) was recently shown to potently inhibit GsdmD, but the in-vivo efficacy and …

Modulation of host lipid membrane elasticity and dipole potential induced by bacterial membrane vesicles

A Prince, A Tiwari, T Mandal, K Sharma, N Kanik… - bioRxiv, 2019 - biorxiv.org
Bacterial membrane vesicles (MVs) facilitate long-distance delivery of virulence factors
crucial for pathogenicity. The entry and trafficking mechanisms of virulence factors inside host …

[PDF][PDF] Interplay of bacterial membrane vesicle and host lipid

A Prince, A Tiwari, T Mandal, K Sharma, N Kanik… - lipids, 2014 - academia.edu
Bacterial membrane vesicles (MVs) facilitate long-distance delivery of virulence factors
crucial for pathogenicity. The entry and trafficking mechanisms of virulence factors inside host …