User profiles for Ashutosh Prince
Ashutosh PrincePostdoctoral Scholar, Cleveland State University Verified email at csuohio.edu Cited by 188 |
[HTML][HTML] Lipid-II independent antimicrobial mechanism of nisin depends on its crowding and degree of oligomerization
Nisin inhibits bacterial growth by generating pores in cell membrane and interrupting cell-wall
biosynthesis through specific lipid II interaction. However, the role of the hinge region and …
biosynthesis through specific lipid II interaction. However, the role of the hinge region and …
Attenuation of neuroblastoma cell growth by nisin is mediated by modulation of phase behavior and enhanced cell membrane fluidity
Antimicrobial peptides have been attracting significant attention as potential anti-cancer
therapeutic agents in recent times. Yet most antimicrobial peptides seem to possess cytotoxic …
therapeutic agents in recent times. Yet most antimicrobial peptides seem to possess cytotoxic …
Passive membrane penetration by ZnO nanoparticles is driven by the interplay of electrostatic and phase boundary conditions
The internalization of nanoparticles through the biological membrane is of immense importance
for biomedical applications. A fundamental understanding of the lipid specificity and the …
for biomedical applications. A fundamental understanding of the lipid specificity and the …
Perturbations of Cellular Model Membranes Induced by Membrane Interacting Model Peptide, Protein and Proteoliposome
A Prince - 2020 - ethesis.nitrkl.ac.in
The cell membrane interface constantly encounters a highly crowded environment of
membrane interacting biomolecular entities present, extracellularly and intracellularly, that both …
membrane interacting biomolecular entities present, extracellularly and intracellularly, that both …
[PDF][PDF] Impavido attenuates inflammation, reduces atherosclerosis, and alters gut microbiota in hyperlipidemic mice
Impavido (Miltefosine) is an FDA-approved drug for treating leishmaniasis and primary amebic
meningoencephalitis. We have shown previously that Miltefosine increased cholesterol …
meningoencephalitis. We have shown previously that Miltefosine increased cholesterol …
Osteoblast‐derived giant plasma membrane vesicles induce osteogenic differentiation of human mesenchymal stem cells
Exosome‐mediated differentiation of human mesenchymal stem cell (hMSC) has opened
up a new possibility for the directed osteogenic differentiation of hMSC by the osteoblast‐…
up a new possibility for the directed osteogenic differentiation of hMSC by the osteoblast‐…
Corrigendum: lipid-II independent antimicrobial mechanism of nisin depends on its crowding and degree of oligomerization
… Ashutosh Prince, Padmani Sandhu, Pankaj Ror, Eva Dash, Shingarika Sharma,
Manoranjan Arakha, Suman Jha, Yusuf Akhter, and Mohammed Saleem …
Manoranjan Arakha, Suman Jha, Yusuf Akhter, and Mohammed Saleem …
Disulfiram reduces atherosclerosis and enhances efferocytosis, autophagy, and atheroprotective gut microbiota in hyperlipidemic mice
CA Traughber, K Timinski, A Prince, N Bhandari… - bioRxiv, 2023 - ncbi.nlm.nih.gov
Pyroptosis executor Gasdermin (GsdmD) promotes atherosclerosis in mice and humans.
Disulfiram (DSF) was recently shown to potently inhibit GsdmD, but the in-vivo efficacy and …
Disulfiram (DSF) was recently shown to potently inhibit GsdmD, but the in-vivo efficacy and …
Modulation of host lipid membrane elasticity and dipole potential induced by bacterial membrane vesicles
Bacterial membrane vesicles (MVs) facilitate long-distance delivery of virulence factors
crucial for pathogenicity. The entry and trafficking mechanisms of virulence factors inside host …
crucial for pathogenicity. The entry and trafficking mechanisms of virulence factors inside host …
[PDF][PDF] Interplay of bacterial membrane vesicle and host lipid
Bacterial membrane vesicles (MVs) facilitate long-distance delivery of virulence factors
crucial for pathogenicity. The entry and trafficking mechanisms of virulence factors inside host …
crucial for pathogenicity. The entry and trafficking mechanisms of virulence factors inside host …