Copy number variants (CNVs) have been strongly implicated in the genetic etiology of
schizophrenia (SCZ). However, genome-wide investigation of the contribution of CNV to risk has …

Recurrent microdeletions and microduplications of a 600-kb genomic region of chromosome
16p11.2 have been implicated in childhood-onset developmental disorders 1 , 2 , 3 . We …

Objective. —The A1 allele of theTaqI polymorphism of the dopamine D 2 receptor (DRD2)
gene has been earlier reported to occur in 69% of alcoholics, compared with 20% of controls. …

We report the identification of a recurrent, 520-kb 16p12.1 microdeletion associated with
childhood developmental delay. The microdeletion was detected in 20 of 11,873 cases …

A simple test of smooth-pursuit eye movements disclosed a striking association between
deviant eye tracking and clinically diagnosed schizophrenia. A high proportion of the …

Rare copy number variants (CNVs) have a prominent role in the aetiology of schizophrenia
and other neuropsychiatric disorders 1 . Substantial risk for schizophrenia is conferred by …

While it is known that rare copy-number variants (CNVs) contribute to risk for some neuropsychiatric
disorders, the role of CNVs in bipolar disorder is unclear. Here, we reasoned that a …

Recent studies have established an important role for rare genomic deletions and duplications
in the etiology of schizophrenia. This research suggests that the genetic architecture of …

Increasing interest in the role of the frontal lobe in relation to psychiatric and neurologic
disorders has popularized tests of frontal function. One of these is the antisaccade task, in which …

Eye tracking dysfunction (ETD) has been found in large numbers of schizophrenia patients
and their first-degree relatives. Because of the many replications of the central findings, ETD …