User profiles for Rochelle L. Coulson

Rochelle Coulson

Stanford University
Verified email at stanford.edu
Cited by 734

R-loop formation at Snord116 mediates topotecan inhibition of Ube3a-antisense and allele-specific chromatin decondensation

WT Powell, RL Coulson, ML Gonzales… - Proceedings of the …, 2013 - National Acad Sciences
Prader–Willi syndrome (PWS) and Angelman syndrome (AS) are oppositely imprinted
autism-spectrum disorders with known genetic bases, but complex epigenetic mechanisms …

A Prader–Willi locus lncRNA cloud modulates diurnal genes and energy expenditure

WT Powell, RL Coulson, FK Crary… - Human molecular …, 2013 - academic.oup.com
Prader–Willi syndrome (PWS), a genetic disorder of obesity, intellectual disability and sleep
abnormalities, is caused by loss of non-coding RNAs on paternal chromosome 15q11-q13. …

[PDF][PDF] Cumulative impact of polychlorinated biphenyl and large chromosomal duplications on DNA methylation, chromatin, and expression of autism candidate genes

KW Dunaway, MS Islam, RL Coulson, SJ Lopez… - Cell reports, 2016 - cell.com
Rare variants enriched for functions in chromatin regulation and neuronal synapses have
been linked to autism. How chromatin and DNA methylation interact with environmental …

Placental DNA methylation levels at CYP2E1 and IRS2 are associated with child outcome in a prospective autism study

…, DH Yasui, R Marathe, RL Coulson… - Human molecular …, 2019 - academic.oup.com
DNA methylation acts at the interface of genetic and environmental factors relevant for autism
spectrum disorder (ASD). Placenta, normally discarded at birth, is a potentially rich source …

Cognitive deficits in the Snord116 deletion mouse model for Prader-Willi syndrome

…, NA Copping, B Onaga, MC Pride, RL Coulson… - Neurobiology of learning …, 2019 - Elsevier
Prader-Willi syndrome (PWS) is an imprinted neurodevelopmental disease caused by a loss
of paternal genes on chromosome 15q11-q13. It is characterized by cognitive impairments, …

[HTML][HTML] Snord116-dependent diurnal rhythm of DNA methylation in mouse cortex

RL Coulson, DH Yasui, KW Dunaway, BI Laufer… - Nature …, 2018 - nature.com
Rhythmic oscillations of physiological processes depend on integrating the circadian clock
and diurnal environment. DNA methylation is epigenetically responsive to daily rhythms, as a …

[HTML][HTML] Sleep deficiency as a driver of cellular stress and damage in neurological disorders

RL Coulson, P Mourrain, GX Wang - Sleep medicine reviews, 2022 - Elsevier
Neurological disorders encompass an extremely broad range of conditions, including those
that present early in development and those that progress slowly or manifest with advanced …

[HTML][HTML] Genome-Wide Association Mapping in Dogs Enables Identification of the Homeobox Gene, NKX2-8, as a Genetic Component of Neural Tube Defects in …

…, AG Bassuk, PJ Ferguson, M Aguilar, RL Coulson… - PLoS …, 2013 - journals.plos.org
Neural tube defects (NTDs) is a general term for central nervous system malformations
secondary to a failure of closure or development of the neural tube. The resulting pathologies …

Experience-dependent neuroplasticity of the developing hypothalamus: integrative epigenomic approaches

…, BI Laufer, KW Dunaway, CE Mordaunt, RL Coulson… - Epigenetics, 2018 - Taylor & Francis
Augmented maternal care during the first postnatal week promotes life-long stress resilience
and improved memory compared with the outcome of routine rearing conditions. Recent …

Prader–Willi locus Snord116 RNA processing requires an active endogenous allele and neuron-specific splicing by Rbfox3/NeuN

RL Coulson, WT Powell, DH Yasui… - Human molecular …, 2018 - academic.oup.com
Prader–Willi syndrome (PWS), an imprinted neurodevelopmental disorder characterized by
metabolic, sleep and neuropsychiatric features, is caused by the loss of paternal SNORD116…