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Capturing a crucial ‘disorder-to-order transition’ at the heart of the coronavirus molecular pathology – triggered by highly persistent, interchangeable salt-bridges

View ORCID ProfileSourav Roy, View ORCID ProfilePrithwi Ghosh, View ORCID ProfileAbhirup Bandyopadhyay, View ORCID ProfileSankar Basu
doi: https://doi.org/10.1101/2021.12.29.474439
Sourav Roy
1Dept of Microbiology and Immunology, Brody School of Medicine, East Carolina University, Greenville, NC, 27834, USA
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Prithwi Ghosh
2Department of Botany, Narajole Raj College, Narajole, Paschim Medinipur, West Bengal, India
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Abhirup Bandyopadhyay
3Theoretical Neurosciences Group, Institute De Neurosciences Des Systems, Aix-Marseille University, France
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Sankar Basu
4Department of Microbiology, Asutosh College (affiliated to University of Calcutta), 92, Shyama Prasad Mukherjee Rd, Bhowanipore, Kolkata, West Bengal 700026, India
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Posted December 29, 2021.
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Capturing a crucial ‘disorder-to-order transition’ at the heart of the coronavirus molecular pathology – triggered by highly persistent, interchangeable salt-bridges
Sourav Roy, Prithwi Ghosh, Abhirup Bandyopadhyay, Sankar Basu
bioRxiv 2021.12.29.474439; doi: https://doi.org/10.1101/2021.12.29.474439
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Capturing a crucial ‘disorder-to-order transition’ at the heart of the coronavirus molecular pathology – triggered by highly persistent, interchangeable salt-bridges
Sourav Roy, Prithwi Ghosh, Abhirup Bandyopadhyay, Sankar Basu
bioRxiv 2021.12.29.474439; doi: https://doi.org/10.1101/2021.12.29.474439

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